Stimulant du système nerveux central, la caféine est un alcaloïde issu du café que l’on retrouve également dans le thé et certaines boissons.
Est-il bon ou mauvais de consommer de la caféine?
Jugez-en par vous même.
- En plus d’augmenter la progestérone dans le sang, la caféine protège contre certains types de cancer causés par les radiations, les produits chimiques, les virus et l’oestrogène;
- Plusieurs études démontrent que le café, le thé et la caféine ont un effet protecteur contre le cancer du sein;
- Les buveurs de café sont moins touchés par les maladies affectant la glande thyroïde, y compris le cancer;
- La caféine protège le foie contre les toxines, y compris l’alcool et l’acétaminophène (Tylenol), les enzymes sériques et d’autres dommages;
- Les buveurs de café ont un taux inférieur de cadmium dans les tissus dû à l’ébullition qui élimine les métaux lourds contenus dans l’eau;
- La caféine réduit l’agrégation plaquettaire et contribue à l’absorption de la sérotonine;
- Le café pris pendant un repas aide à prévenir une surabsorption de fer par l’organisme;
- Le café (ou la caféine) réduit les risques de développer la maladie de Parkinson;
- La caféine arrête la production de radicaux libres par l’inhibition de la xanthine oxydase;
- La caféine aide à réduire la production de potassium sérique après l’exercice, stabilise les plaquettes et aide à réduire la production de thromboxane;
- Le café fournit des quantités importantes de magnésium et d’autres nutriments, dont la vitamine B1;
- Tout comme la niacine, la caféine inhibe l’apoptose (auto-destruction des cellules induite par le stress) sans interférer avec le renouvèlement cellulaire normal;
À NOTER : Bien qu’il ait été démontré qu’un retard de croissance prénatal pourrait être causé par l’ingestion d’une trop grande quantité de caféine, cet effet peut être renversé en ajoutant du sucre à son alimentation.
Il ne vous reste plus qu’à vous préparer un bon café!
À votre santé!
Sources et références:
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Pharmacol Biochem Behav 2000 May;66(1):39-45. Caffeine-induced increases in the brain and plasma concentrations of neuroactive steroids in the rat. Concas A, Porcu P, Sogliano C, Serra M, Purdy RH, Biggio G.
Cancer Res 1998 Sep 15;58(18):4096-101. Inhibition of lung carcinogenesis by black tea in Fischer rats treated with a tobacco-specific carcinogen: caffeine as an important constituent. Chung FL, Wang M, Rivenson A, Iatropoulos MJ, Reinhardt JC, Pittman B, Ho CT, Amin SG.
Cancer Lett 1991 Mar;56(3):245-50. Inhibition by caffeine of ovarian hormone-induced mammary gland tumorigenesis in female GR mice. VanderPloeg LC, Welsch CW.
Breast Cancer Res Treat 1991 Nov;19(3):269-75. Caffeine inhibits development of benign mammary gland tumors in carcinogen-treated female Sprague-Dawley rats. Wolfrom DM, Rao AR, Welsch CW.
Cancer 1985 Oct 15;56(8):1977-81. The inhibitory effect of caffeine on hormone-induced rat breast cancer. Petrek JA, Sandberg WA, Cole MN, Silberman MS, Collins DC.
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J Environ Pathol Toxicol Oncol 1992; 11(3):177-89. Caffeine, theophylline, theobromine, and developmental growth of the mouse mammary gland. VanderPloeg LC, Wolfrom DM, Rao AR, Braselton WE, Welsch CW.
J Environ Pathol Toxicol Oncol 1994;13(2):81-8. Enhancement by caffeine of mammary gland lobulo-alveolar development in mice: a function of increased corticosterone. Welsch CW, VanderPloeg LC. Previously we have reported that the stimulatory effect of caffeine on lobulo-alveolar development in the mammary glands of female Balb/c mice is not due to a direct action of the drug on the mammary gland but appears to be due to a caffeine-induced alteration of a yet to be defined systemic physiological process (VanderPloeg et al., J Environ Pathol Toxicol Oncol 11:177-189, 1992). “In the present study, we administered caffeine (via the drinking water, 500 mg/L) to ovariectomized, estrogen- and progesterone-treated Balb/c mice. After 30 days of caffeine treatment, a significant (p < 0.001) enhancement of lobulo-alveolar development in the mammary glands of the hormone-treated mice, compared with hormone treated control mice, was observed.”
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Gen Pharmacol 1996 Jan;27(1):167-70 The influence of antagonists of poly(ADP-ribose) metabolism on acetaminophen hepatotoxicity. Kroger H, Ehrlich W, Klewer M, Gratz R, Dietrich A, Miesel R.
Carcinogenesis 1998 Aug;19(8):1369-75. The coffee-specific diterpenes cafestol and kahweol protect against aflatoxin B1-induced genotoxicity through a dual mechanism. Cavin C, Holzhauser D, Constable A, Huggett AC, Schilter B.
Am J Epidemiol 1994 Apr 1;139(7):723-7. Coffee and serum gamma-glutamyltransferase: a study of self-defense officials in Japan. Kono S, Shinchi K, Imanishi K, Todoroki I, Hatsuse K.
Carcinogenesis 1996 Nov;17(11):2377-84 Placental glutathione S-transferase (GST-P) induction as a potential mechanism for the anti-carcinogenic effect of the coffee-specific components cafestol and kahweol. Schilter B, Perrin I, Cavin C, Huggett AC
J Nutr 1999 Jul;129(7):1361-7. Teas and other beverages suppress D-galactosamine-induced liver injury in rats. Sugiyama K, He P, Wada S, Saeki S
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